Campbell AR, Satran D, Zenovich AG, Campbell KM, Espel JC, Arndt TL, Poulose AK, Boisjolie CR, Juusola K, Bart BA, Henry TD. Enhanced external counterpulsation improves systolic blood pressure in patients with refractory angina. Am Heart J. 2008 Dec;156(6):1217-22. doi: 10.1016/j.ahj.2008.07.024. Epub 2008 Oct 5. PMID: 19033023.

BACKGROUND:

Enhanced external counterpulsation (EECP) is a noninvasive treatment of patients with refractory angina. The immediate hemodynamic effects of EECP are similar to intra-aortic balloon pump counterpulsation, but EECP’s effects on standard blood pressure measurements during and after treatment are unknown.

METHODS:

We evaluated systolic blood pressure (SBP) and diastolic blood pressure (DBP) for 108 consecutive patients undergoing EECP. Baseline SBP, DBP, and heart rate were compared for each patient before and after each EECP session, at the end of the course of EECP, and 6 weeks after the final EECP session.

RESULTS:

One hundred eight patients (mean age 66.4 +/- 11.2 years, 81% male) completed 36.5 +/- 5.1 EECP sessions per patient. Overall, based on 3,586 individual readings, EECP resulted in a decrease in mean SBP of 1.1 +/- 15.3 mm Hg at the end of each EECP session (P < .001), 6.4 +/- 18.2 mm Hg at the end the course of EECP (P < .001), and 3.7 +/- 17.8 mm Hg 6 weeks after the final EECP session (P = .07), with no significant change in DBP or heart rate. Stratifying by baseline SBP, a differential response was demonstrated: SBP increased in the 2 lowest strata (<100 mm Hg and 101-110 mm Hg) and decreased in the remaining strata (P < .001). Stratified differences were sustained after individual EECP sessions, at the end of the course of EECP, and 6 weeks after the final EECP session and were independent of changes in cardiovascular medications.

CONCLUSIONS:

Enhanced external counterpulsation improved SBP in patients with refractory angina. On average, EECP decreased SBP during treatment and follow-up; but for patients with low baseline SBP (<110 mm Hg), EECP increased SBP. The improvements in SBP may contribute to the clinical benefit of EECP.

Source From : https://bit.ly/3ZscxQ9

「體外反搏治療有效改善難治性心絞痛患者的收縮壓」

 背景

體外反搏治療(EECP)是一種非侵入性治療方法,用於治療難治性心絞痛患者。EECP的即時血液動力學效應與主動脈內氣球幫浦反搏類似,但EECP對治療過程中和治療後的標準血壓測量的影響尚不清楚。

方法

我們評估了108名接受EECP治療的連續患者的收縮壓和舒張壓。對於每位患者,在每次EECP療程前後、EECP療程結束時以及最後一次EECP療程後的6週,比較了基線收縮壓、舒張壓和心率。

結果

共有108名患者完成了36.5次的EECP療程。總體而言,根據3586個單獨的讀數,每次EECP療程結束時平均收縮壓下降了1.1毫米汞,EECP療程結束時下降了6.4毫米汞柱,最後一次EECP療程後的6週下降了3.7毫米汞柱,舒張壓和心率沒有顯著變化。根據基線收縮壓的分層分析,顯示出不同的反應:在最低的兩個分層中收縮壓增加,而在其餘分層中下降。分層差異在個別EECP療程後、EECP療程結束時以及最後一次EECP療程後的6週持續存在,並且與心血管藥物的變化無關。

結論

體外反搏治療改善了難治性心絞痛患者的收縮壓。平均而言,在治療和隨訪過程中,EECP會降低收縮壓;但對於基線收縮壓較低的患者,EECP會增加收縮壓。收縮壓的改善可能有助於EECP的臨床效益。

EECP May be Useful as Adjuvant Therapy to Reduce Central Blood Pressure and Aortic Pulse Pressure

體外反搏治療可能作為輔助療法,能降低中心血壓和主動脈脈壓

ECP(Enhanced External Counterpulsation)體外反搏治療是一種安全、非入侵和具有臨床實證支持的療法,旨在改善心血管狀態和整體健康。該療法使用先進的輔助儀器,通過泵動腿部的壓力,協助血液從下肢回流到心臟,並進一步流向各個器官,以促進血液循環。

ECP治療的主要原理是在心臟收縮之前的舒張期間,通過輔助儀器對腿部施加壓力,產生一種反搏效應。這種反搏效應可以增加主動脈的壓力,從而增加冠狀動脈灌注壓,改善心肌供血。同時,在心臟舒張期間,施加的壓力減少,有助於減輕心臟負荷,提高心臟的充盈和排血功能。這種週期性的反搏和減壓可以增加冠狀動脈血流,改善心臟和全身各個器官的血液供應。

ECP治療的好處有多方面。首先,它是一種非侵入性的治療方法,不需要進行手術或使用藥物,減少了相關風險和副作用。其次,體外反搏治療通常被認為是安全的,並且可以在診所或醫院中進行,無需住院。此外,這種治療方法已被廣泛研究和臨床實踐,具有豐富的臨床實證支持。

ECP治療對於心血管疾病和其他相關疾病的改善效果已被廣泛證實。它被廣泛應用於治療心絞痛(心絞痛是由於冠狀動脈供血不足引起的胸痛)、心衰竭和冠狀動脈疾病等疾病。此外,ECP治療還被用於改善運動耐力、減輕疲勞和提高生活質量。

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